First, read the original post at http://www.stephanieaustin.com/blog/sat-oct-25-2014.
This reader then asks: Where can I find independence controlled studies, or scientific research that supports these claims? That are not related to the author, nor products.
Coach Stephanie: Which claims are you referring to? Fat metabolism and toxin storage? That the body can heal itself? That diets don't work? There are no products to my program.
Reader: Research that Calorie and Exercise ultimately cause an increase in toxic density. That toxic overload triggers the body to create fat? How removing toxins can actually create lean muscle. Not trying to argue, but I'm interested and like you I'm into the science of the claims....
Coach Stephanie: You betcha! I don't see it as argument at all. I LOVE research and studies, as I'm sure you knew already. :0) Fat metabolism and lipid storage of toxins is textbook stuff, which I will be happy to go back to. I have many of my school books that I can refer you to. But there are many studies as well. Let me gather a few for you when I get to my computer this afternoon - I'm on my phone at the moment. I've not been online as much of late, so I apologize for my late reply. In brief, the gist being that when the fat cells are metabolized during calorie counting type diets - which I don't promote in most cases - the released toxins create an overly-acidic environment which requires alkaline-forming agents to assist. When they aren't present, the body will send signals which essentially create more fat to protect the body from the overload. I can tell you like the Science stuff, so I will give you some actual studies to review for yourself. I have an entire section on fat metabolism within my Restoring Wellness program too. ANyhow.... I'll post later today. Thanks for the opportunity!
Coach Stephanie: Like I’d mentioned above, fat metabolism without proper detoxification will reintroduce toxins that were ‘safely stored as excess’ within fat cells, and put them back into the system. This leads to an overly acidic environment; depressed metabolism due to decreased thyroid function; an increase in inflammatory-promoting hormones; decreased liver function; reproductive issues; increased weight gain… the list of consequences go on quite frankly. You can see why it is taught within and throughout the 6-week program.
There are many references to this though, such as the EPA’s 1982 program called “National Human Adipose Tissue Survey” whereby they analyzed “fat samples from cadavers from across the country, looking for the types of toxins that accumulate in human fat. Four industrial solvents and one dioxin were found in 100 percent of the fat samples. Another nine chemicals, including three more dioxins and one furan were found in more than 90 percent of the fat samples. In general, 83 percent of the fat samples contained PCBs.” You can read more on it here: http://cfpub.epa.gov/ncea/cfm/recordisplay.cfm?deid=55204
We are constantly exposed to these toxins in our own lifestyles but they are also found in the soil and within the animals and animal products we eat. Aside from overly acidifying the body’s blood when these toxins are released, th accumulation of these toxins can also depress the function of thyroid which greatly affects metabolism on its own. The Physical Activity Sciences Laboratory in Canada did a study on this here:http://toxsci.oxfordjournals.org/content/67/1/46.full where it showed: “Organochlorine compounds are released from body fat into the bloodstream during weight loss. Because these compounds may impair thyroid status, which is implicated in the control of resting metabolic rate (RMR), the aim of this study was to determine if the augmentation in plasma organochlorine concentrations might be associated with the decrease in serum T(3) concentration and RMR observed in response to body weight loss. Plasma organochlorine concentrations, serum T(3) concentration, and RMR were measured before and after weight loss in 16 obese men who followed a nonmacronutrient-specific energy-restricted diet for 15 weeks. As expected, a significant decrease in serum T(3) concentration and RMR was observed after the program, whereas concentrations of most detected organochlorines were significantly increased. Changes in organochlorine concentrations were negatively associated with changes in serum T(3) concentration (significantly for p,p’-DDT, HCB, Aroclor 1260, PCB 28, PCB 99, PCB 118, and PCB 170) and with changes in RMR adjusted for weight loss (significantly for HCB and PCB 156). In conclusion, organochlorines released in plasma during weight loss are associated with the documented decrease in serum T(3) concentration and RMR. Further studies are needed to verify whether these findings are causally related.”
Also, the CDC’s “National Health and Nutrition Examination Survey” (NHANES) showed “serum concentrations of six persistent organic pollutants were significantly correlated with weight change, according to Duk-Hee Lee, MD, PhD, of Kyungpook National University in Daegu, Korea, and colleagues.” The data came from 1099 adults. The CDC also released a report showing chemicals stored in fatty tissue included bisphenal A and flame retardants, such as those used in household building materials. Lee states: “"researchers and clinicians need to consider lipophilic xenobiotics such as persistent organic pollutants that bioaccumulate in adipose tissue as well as obesity itself when they study or manage obesity issues because such xenobiotics may work against what we generally expect from weight loss or gain.”
Also in this study, though not specific to your question, showed the average newborn has “287 chemicals in the umbilical cord, 217 of which are neurotoxic.”
Additional info you might find interesting on fatty tissue, inflammation and subsequent weight gain: “The significance of inflammatory responses elicited via secretion of adipose tissue-derived (WAT) cytokines relates to the fact that their production and secretion is increased in obese individuals. There is a growing body of evidence that demonstrates a direct link between the changes in adipose tissue function in obesity and the development of type 2 diabetes and the metabolic syndrome. One key change in adipose tissue during obesity is an increase in the percentage of macrophages resident within the tissue. Macrophages are a primary source of pro-inflammatory cytokines secreted by adipose tissue. The primary adipokine responsible for this infiltration is monocyte chemotactic protein-1 (MCP-1).
As the level of macrophages increases in adipose tissue the level of pro-inflammatory cytokine secretion by the tissue increases. Circulating levels of both TNF-α and IL-6 increase as adipose tissue expands in obesity and these changes are directly correlated with insulin resistance and the development of type 2 diabetes.” From: http://lib.dr.iastate.edu/cgi/viewcontent.cgi...
Here are a few more interesting studies:
Body weight loss increases plasma and adipose tissue concentrations of potentially toxic pollutants in obese individuals.http://www.ncbi.nlm.nih.gov/m/pubmed/11093288/
“Wayne State University researchers examining how toxicity of fatty acids links obesity and diabetes - As people become obese, their adipose tissue, which stores energy from food, loses its ability to do so, releasing toxic lipids, or free fatty acids (FFAs), which make their way to muscles and the liver. The FFAs then interfere with insulin's ability to promote the use of glucose as a fuel by cells in the muscle and liver. As a result, the pancreas is stimulated to produce more insulin, but diabetes can occur if the pancreas is unable to meet the higher demand. Some 20 million people in the United States suffer from type 2 diabetes and its complications.
Anyhow, the program teaches how to reach ones optimal weight while optimizing digestion, increasing and improving elimination, and clearing out the blood. I hope that answered some of your curiosities.